ALS Awareness Month Shows
Progress by MDA Researchers
TUCSON, Ariz., May 1, 2005 — Research funded by the Muscular
Dystrophy Association during the past year has identified potential
drug treatments for ALS (amyotrophic
lateral sclerosis, or Lou Gehrig’s disease) and uncovered
new triggers and risk factors for the disease.
May marks the 14th annual national ALS Awareness Month.
“MDA has led the fight to defeat ALS for 50 years and
funds more ALS research than any other voluntary health organization in the world,”
MDA CEO & President Robert Ross said. “As we observe
ALS Awareness Month this year, we remain deeply committed to
the pursuit of more effective treatments or a cure for this
devastating disease.”
MDA supports investigations around the world into ALS, which
attacks muscle-controlling nerve cells (motor neurons) and typically
leads to paralysis and death within two to five years of diagnosis.
More than 30,000 American adults have ALS, and the cause is
still unknown.
Highlights of MDA’s research progress in ALS over the
past year include:
ALS TRIGGERS AND RISK FACTORS
A research group that had MDA support concluded that activation
of dormant viruses in ALS patients’ DNA could play a role
in development of the disease. The conclusion is based on elevated
levels of an enzyme called reverse transcriptase, used by retroviruses
when they replicate, in the blood serum of ALS patients and
their relatives.
MDA grantees identified as ALS risk factors variants in the
gene for Hfe, which result in iron-related toxicity; and in
the gene for peripherin, resulting in obstructive clumps of
cellular material.
ALS TREATMENTS
Several clinical trials of compounds to treat ALS are taking
place now or will be in the near future at MDA/ALS centers across
the country. Among them are studies of:
- a new type of antioxidant called AEOL 10150
- the antioxidant coenzyme Q10
- the neurotrophic (nerve-nourishing) factor insulin-like growth
factor 1 (Myotrophin)
- the antibiotic ceftriaxone, which has shown promise in removing
potentially toxic glutamate from the area around nerve cells
- the antibiotic minocycline, which shows promise in reducing
inflammation and keeping cells from activating a “cell
death” program
- glatiramer acetate (Copaxone), which is used in multiple sclerosis
and changes immune system activity
- a new class of drugs called HDAC inhibitors, which may change
the genetic program launched by nerve cells in response to stress.
For more about MDA’s ALS Division and ALS Awareness Month
activities, visit www.als-mda.org. |